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Instructions

Student presentations must have a faculty sponsor.

Abstracts must include a title and a description of the research, scholarship, or creative work. The description should be 150-225 words in length and constructed in a format or style appropriate for the presenter’s discipline.

The following points should be addressed within the selected format or style for the abstract:

  • A clear statement of the problem or question you pursued, or the scholarly goal or creative theme achieved in your work.
  • A brief comment about the significance or uniqueness of the work.
  • A clear description of the methods used to achieve the purpose or goals for the work.
  • A statement of the conclusions, results, outcomes, or recommendations, or if the work is still in progress, the results you expect to report at the event.

Presenter photographs should be head and shoulder shots comparable to passport photos.

Additional Information

More information is available at carthage.edu/celebration-scholars/. The following are members of the Research, Scholarship, and Creativity Committee who are eager to listen to ideas and answer questions:

  • Jun Wang
  • Kim Instenes
  • John Kirk
  • Nora Nickels
  • Andrew Pustina
  • James Ripley

Apoptosis: A Target for Anticancer Therapy

Name: Claire Pfeffer
Major: Biology
Hometown: Oregon, WI
Faculty Sponsor: Amareshwar Singh
Other Sponsors:  
Type of research: Independent research

Abstract

Apoptosis, the cell’s natural mechanism for death, is a promising target for anticancer therapy. Both the intrinsic and extrinsic pathways use caspases to carry out apoptosis through the cleavage of hundreds of proteins. In cancer, the apoptotic pathway is typically inhibited through a wide variety of means including overexpression of antiapoptotic proteins and under-expression of proapoptotic proteins. Many of these changes cause intrinsic resistance to the most common anticancer therapy, chemotherapy. Promising new anticancer therapies are plant-derived compounds that exhibit anticancer activity through activating the apoptotic pathway.

Poster file

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